acetylcholine chloride accl Search Results


cholinergic agonist methacholine  (Methapharm Inc)
90
Methapharm Inc cholinergic agonist methacholine
Cholinergic Agonist Methacholine, supplied by Methapharm Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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vesicular acetylcholine transporter protein  (Santa Cruz Biotechnology)
93
Santa Cruz Biotechnology vesicular acetylcholine transporter protein
Vesicular Acetylcholine Transporter Protein, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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3h acetylcholine iodide  (Revvity)
90
Revvity 3h acetylcholine iodide
3h Acetylcholine Iodide, supplied by Revvity, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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gene exp ache hs00241307 m1  (Thermo Fisher)
96
Thermo Fisher gene exp ache hs00241307 m1
PGC-1 α _hMPCs induce expression of genes related to vascularization, mitochondrial, and neuronal activation and enhance the contractility at early and midterm time points after TA crush injury during regeneration. RTPCR analysis confirmed the sustained overexpression of hPGC-1 α and the signalling-deficient hD2R genes at the crush injury site over time. Relative VEGF-A, COXIV, and <t>ACHE</t> gene levels were enhanced in the corresponding samples, compared to control GFP_hMPC at (a) early, (b) midterm, and (c) late time points of regeneration. (d) PGC-1 α overexpression led to increased TA contractile force at early and midterm time points. Native TA muscle (TA nat) was used as control, and the contraction force was set to 100%. Forces of injured muscles were calculated relatively as the percentage of maximum. VEGF-A: vascular endothelial growth factor-a, COXIV: cytochrome c oxidase subunit 4, ACHE: acetylcholine esterase. ∗ p < 0.05, ∗∗ p < 0.01, ∗∗∗ p < 0.001, and ∗∗∗∗ p < 0.0001.
Gene Exp Ache Hs00241307 M1, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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gene exp ache hs00241307 m1 - by Bioz Stars, 2026-05
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acetyl choline chloride  (Santa Cruz Biotechnology)
93
Santa Cruz Biotechnology acetyl choline chloride
PGC-1 α _hMPCs induce expression of genes related to vascularization, mitochondrial, and neuronal activation and enhance the contractility at early and midterm time points after TA crush injury during regeneration. RTPCR analysis confirmed the sustained overexpression of hPGC-1 α and the signalling-deficient hD2R genes at the crush injury site over time. Relative VEGF-A, COXIV, and <t>ACHE</t> gene levels were enhanced in the corresponding samples, compared to control GFP_hMPC at (a) early, (b) midterm, and (c) late time points of regeneration. (d) PGC-1 α overexpression led to increased TA contractile force at early and midterm time points. Native TA muscle (TA nat) was used as control, and the contraction force was set to 100%. Forces of injured muscles were calculated relatively as the percentage of maximum. VEGF-A: vascular endothelial growth factor-a, COXIV: cytochrome c oxidase subunit 4, ACHE: acetylcholine esterase. ∗ p < 0.05, ∗∗ p < 0.01, ∗∗∗ p < 0.001, and ∗∗∗∗ p < 0.0001.
Acetyl Choline Chloride, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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acetyl-3h] acetylcholine iodide (ach)  (DuPont de Nemours)
90
DuPont de Nemours acetyl-3h] acetylcholine iodide (ach)
PGC-1 α _hMPCs induce expression of genes related to vascularization, mitochondrial, and neuronal activation and enhance the contractility at early and midterm time points after TA crush injury during regeneration. RTPCR analysis confirmed the sustained overexpression of hPGC-1 α and the signalling-deficient hD2R genes at the crush injury site over time. Relative VEGF-A, COXIV, and <t>ACHE</t> gene levels were enhanced in the corresponding samples, compared to control GFP_hMPC at (a) early, (b) midterm, and (c) late time points of regeneration. (d) PGC-1 α overexpression led to increased TA contractile force at early and midterm time points. Native TA muscle (TA nat) was used as control, and the contraction force was set to 100%. Forces of injured muscles were calculated relatively as the percentage of maximum. VEGF-A: vascular endothelial growth factor-a, COXIV: cytochrome c oxidase subunit 4, ACHE: acetylcholine esterase. ∗ p < 0.05, ∗∗ p < 0.01, ∗∗∗ p < 0.001, and ∗∗∗∗ p < 0.0001.
Acetyl 3h] Acetylcholine Iodide (Ach), supplied by DuPont de Nemours, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/acetyl-3h] acetylcholine iodide (ach)/product/DuPont de Nemours
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4c]-acetylcholine [acetylcholine iodide (acetyl-ll4c)  (New England Nuclear Corporation)
90
New England Nuclear Corporation 4c]-acetylcholine [acetylcholine iodide (acetyl-ll4c)
PGC-1 α _hMPCs induce expression of genes related to vascularization, mitochondrial, and neuronal activation and enhance the contractility at early and midterm time points after TA crush injury during regeneration. RTPCR analysis confirmed the sustained overexpression of hPGC-1 α and the signalling-deficient hD2R genes at the crush injury site over time. Relative VEGF-A, COXIV, and <t>ACHE</t> gene levels were enhanced in the corresponding samples, compared to control GFP_hMPC at (a) early, (b) midterm, and (c) late time points of regeneration. (d) PGC-1 α overexpression led to increased TA contractile force at early and midterm time points. Native TA muscle (TA nat) was used as control, and the contraction force was set to 100%. Forces of injured muscles were calculated relatively as the percentage of maximum. VEGF-A: vascular endothelial growth factor-a, COXIV: cytochrome c oxidase subunit 4, ACHE: acetylcholine esterase. ∗ p < 0.05, ∗∗ p < 0.01, ∗∗∗ p < 0.001, and ∗∗∗∗ p < 0.0001.
4c] Acetylcholine [Acetylcholine Iodide (Acetyl Ll4c), supplied by New England Nuclear Corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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3h-acetyl] acetylcholine  (Zinsser Analytic)
90
Zinsser Analytic 3h-acetyl] acetylcholine
PGC-1 α _hMPCs induce expression of genes related to vascularization, mitochondrial, and neuronal activation and enhance the contractility at early and midterm time points after TA crush injury during regeneration. RTPCR analysis confirmed the sustained overexpression of hPGC-1 α and the signalling-deficient hD2R genes at the crush injury site over time. Relative VEGF-A, COXIV, and <t>ACHE</t> gene levels were enhanced in the corresponding samples, compared to control GFP_hMPC at (a) early, (b) midterm, and (c) late time points of regeneration. (d) PGC-1 α overexpression led to increased TA contractile force at early and midterm time points. Native TA muscle (TA nat) was used as control, and the contraction force was set to 100%. Forces of injured muscles were calculated relatively as the percentage of maximum. VEGF-A: vascular endothelial growth factor-a, COXIV: cytochrome c oxidase subunit 4, ACHE: acetylcholine esterase. ∗ p < 0.05, ∗∗ p < 0.01, ∗∗∗ p < 0.001, and ∗∗∗∗ p < 0.0001.
3h Acetyl] Acetylcholine, supplied by Zinsser Analytic, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/3h-acetyl] acetylcholine/product/Zinsser Analytic
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3h-acetyl] acetylcholine - by Bioz Stars, 2026-05
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acetylcholine chloride  (Supelco)
N/A
Acetylcholine Chloride
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acetylcholine chloride  (Biosynth Carbosynth)
N/A
Acetylcholine Chloride chemical reference substance
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acetylcholine-d9 chloride  (Toronto Research Chemicals)
N/A
Acetylcholine-d9 Chloride
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Image Search Results


PGC-1 α _hMPCs induce expression of genes related to vascularization, mitochondrial, and neuronal activation and enhance the contractility at early and midterm time points after TA crush injury during regeneration. RTPCR analysis confirmed the sustained overexpression of hPGC-1 α and the signalling-deficient hD2R genes at the crush injury site over time. Relative VEGF-A, COXIV, and ACHE gene levels were enhanced in the corresponding samples, compared to control GFP_hMPC at (a) early, (b) midterm, and (c) late time points of regeneration. (d) PGC-1 α overexpression led to increased TA contractile force at early and midterm time points. Native TA muscle (TA nat) was used as control, and the contraction force was set to 100%. Forces of injured muscles were calculated relatively as the percentage of maximum. VEGF-A: vascular endothelial growth factor-a, COXIV: cytochrome c oxidase subunit 4, ACHE: acetylcholine esterase. ∗ p < 0.05, ∗∗ p < 0.01, ∗∗∗ p < 0.001, and ∗∗∗∗ p < 0.0001.

Journal: Stem Cells International

Article Title: Injected Human Muscle Precursor Cells Overexpressing PGC-1 α Enhance Functional Muscle Regeneration after Trauma

doi: 10.1155/2018/4658503

Figure Lengend Snippet: PGC-1 α _hMPCs induce expression of genes related to vascularization, mitochondrial, and neuronal activation and enhance the contractility at early and midterm time points after TA crush injury during regeneration. RTPCR analysis confirmed the sustained overexpression of hPGC-1 α and the signalling-deficient hD2R genes at the crush injury site over time. Relative VEGF-A, COXIV, and ACHE gene levels were enhanced in the corresponding samples, compared to control GFP_hMPC at (a) early, (b) midterm, and (c) late time points of regeneration. (d) PGC-1 α overexpression led to increased TA contractile force at early and midterm time points. Native TA muscle (TA nat) was used as control, and the contraction force was set to 100%. Forces of injured muscles were calculated relatively as the percentage of maximum. VEGF-A: vascular endothelial growth factor-a, COXIV: cytochrome c oxidase subunit 4, ACHE: acetylcholine esterase. ∗ p < 0.05, ∗∗ p < 0.01, ∗∗∗ p < 0.001, and ∗∗∗∗ p < 0.0001.

Article Snippet: Predesigned primers for human PPARGC-1 (Hs01016719_m1), D2DR (dopamine 2 receptor, Hs00241436_m1), VEGF (vascular endothelial growth factor, Hs00900055_m1), MyH1 (myosin heavy chain 1, Hs00428600_m1), MyH2 (myosin heavy chain 2, Hs00430042_m1), Desmin (Hs00157258_m1), α -actinin (Hs00998100_m1), COXIV (cytochrome c oxidase subunit 4,Hs00971639_m1), vWf (Mm00550375_m1), TNF- α (tumor necrosis factor alpha, Mm00443258_m1), and ACHE (acetyl choline esterase, Hs00241307_m1) were purchased from Life Technologies.

Techniques: Expressing, Activation Assay, Reverse Transcription Polymerase Chain Reaction, Over Expression, Control, Muscles